are sent to the base headquarters, the lymphnodes and thymus, where the decision is made to trigger off the fight. Intricate details are noted about the type, quantity, quality and severity of the invasion before decisions are made about what type of army cell (hymphoyte, eosinophils or monocytes,) or chemicals (antibodies, compliments, local hormones) are asked to go to the site. These are carried through blood stream and a warfare (inflammation) takes place. The white blood cells and the chemical fighters surround the invader, break the bacterial cell wall and dissolve the cell material. In the process the WBCs are themselves destroyed. After all the germs or foreign material are destroyed,s the debris liquifies (pus), and is thrown out and healing begins by filling up the damaged area by the surrounding healthy tissue or scar tissue. Of all the steps involved in this complex immune processes, the most important and sensitive issue is the capacity of the WBCs to sense danger and differentiate foreign cells from own cells. This is a unique and most highly evolved faculty that is specific to a race or an individual. Your immune cells do this by identifying the chemical differences between protein of the foreign cell and your own body cells.

In auto-immunity this capacity to differentiate ‘self from non-self’ is disturbed. The WBCs think that one particular type of self protein is foreign and send messages to gather fighter cells to that particular area and inflammation begins. In ulcerative colitis the cells in the lining of the large intestine are considered foreign and the inflammation (fight) is triggered off. This leads to chronic repeated long-standing fight (a kind of a civil war) in the lining of the large intestine resulting in diarrhoea, blood, mucus, ulcer formation, pain etc.

Stress, psyche and auto-immunity.

The big question that is being asked is why did the WBCs act like this? What made the WBCs do this? What made the WBCs misbehave this way and what made them lose their capacity to